Introduction: Amaurosis fugax is described as a tem-porary loss of vision in one or both eyes which lasts a few seconds or maximum minutes. It is also a symptom that usually is neglected by the patients, with extremely severe consequences. The treatment is targeting the de-termining cause.
Methods: We are presenting a case of a 65-year-old fe-male patient, with venous insuficiency, dyslipidemic, hypertensive who comes to the emergency room after 48 hours of evolution at home of a phlebites episode at the level of the left lower limb accompanied by im-paired function. A thorough anamnesis figured out an episode of amaurosis fugax 72 hours before hospitaliza-tion and the neurological examination at that moment confirmed the supposition diagnosis of right carotidian transient ischeimc attack, but the patient temporised the admission in the hospital.
Results: The complex evaluation objectified the arteri-ovenous affection with the presence of the thrombosis in the venous deep axis with the interest of the posteri-or tibial veins which involved the initiation of the an-ticoagulant therapy, with the significant clinical impro-vement by the reduction of the edema and inflamatory processes, but also with the almost complete mobility resumption of the left lower limb. Considering the polymorphic clinical picture, a pulmonary scintigra-phy was performed and the scintigraphic image show-ed the presence of the pulmonary thromboembolism (intense perfusion disturbances in both lungs).
Conclusions: T he clinical spectrum of the venous thromboembolism varies from asymptomatic cases or with mild symptomatology, to cases which develop hemodynamic instability. The presence of a circum-stantially risk factor could evolve the emergence of the thromboembolic events, with disabling complications or even lethal. The complexity of the etiopathogenic mechanism in thrombembolic disease requires a inter-disciplinary colaboration.